Trenbolone post cycle therapy

It was once commonly used during PCT in the belief it will aid testosterone restoration, however this is flawed due to its mechanism of action. The drug mimics the effects of LH in the body, stimulating the Leydig cells to produce testosterone in the testes . This can be fruitful in rectify existing, or avoiding testicular atrophy on cycle. It will not aid the process of recovery in the post cycle phase however, as the drug will bring about heightened oestrogen levels due to the greater aromatising of the testosterone being produced in the testes , thus bringing about greater inhibition of the HPTA .

Instructions:
Day 1-7: - Clomiphene citrate is used as 50 mg twice daily for full 7 days.
Day 8-37: - Tamoxifene citrate at 20mg per day for full 30 days. - Exemestane at 20mg per day for full 30 days.  HCG 5000 comes in one unit vial, multi-dosed, at 5000IU. The vial is accompanied by 2ml sterile water for mixing. This is what should be done. Draw up the 2ml ampule provided with the HCG and mix it into the powdered bottle. The HCG is now used from day 8 onwards as drawn into insulin needle once per day. It is then used one day on, one day off.

Aromatase inhibitors are the compounds that serve to reduce estradiol levels in blood by eliminating the production of estradiol through binding to and disabling the aromatase enzyme, which is responsible for the conversion (or aromatization) of androgens into estradiol. Suicidal aromatase inhibitors serve to permanently inhibit and disable the aromatase enzyme to which it is bound to. This renders the enzyme inactive forever. The body will eventually manufacture more aromatase enzymes, but the currently-bound enzymes are bound indefinitely, eliminating any risk for estrogen rebound. This is the main difference compared alongside two other major aromatase inhibitors: anastrozole and letrozole, which are non-suicidal aromatase inhibitors that are only bound to the aromatase enzyme for limited time periods. If a non-suicidal aromatase inhibitor is halted too abruptly, the circulating inhibited aromatase enzymes that have not been metabolized out of the body will then become free again, and begin aromatizing androgens into estrogens at an often rapid rate. This is not the case with Exos.

I shared this protocol in the forums i frequent as food for thought and feed back . Boy did i ever get lit up by people saying this cycle is stupid , makes no fucking sense and is complicated for a first time cycle . They say why use letrozole when you don’t know how his E2 levels will react , why use this much test , why start with a long ester then go to a short one , why have both , they said the letro dose is way too strong and will crash the e2 levels and also that its almost impossible to break a pill into that dose also . What are your thoughts on this? you didn’t go into enough depth on why this cycles set up this way , why using letro over something else ? since there is no protocol listed for an alternative and also why using the esters the way you set them up and why front loading with a long ester that apparently wont even kick in for like 4 weeks or more .

The best course of action may be to utilize enough non-hormone steroids, and during the last phase of the cycle, using fast acting substances like testosterone propionate or trenbolone. Oral dianabol may also be effective as it can clear away quickly from the body. Also, during the whole cycle, efforts should be made to keep the levels of estrogen down. This can be done with the help of compounds like formestane and arimidex . Taking non-aromatizable steroids like winstrol and trenbolone is also a good way to keep estrogen levels under control.

Trenbolone post cycle therapy

trenbolone post cycle therapy

I shared this protocol in the forums i frequent as food for thought and feed back . Boy did i ever get lit up by people saying this cycle is stupid , makes no fucking sense and is complicated for a first time cycle . They say why use letrozole when you don’t know how his E2 levels will react , why use this much test , why start with a long ester then go to a short one , why have both , they said the letro dose is way too strong and will crash the e2 levels and also that its almost impossible to break a pill into that dose also . What are your thoughts on this? you didn’t go into enough depth on why this cycles set up this way , why using letro over something else ? since there is no protocol listed for an alternative and also why using the esters the way you set them up and why front loading with a long ester that apparently wont even kick in for like 4 weeks or more .

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